eclampsia, parthenogenesis, preeclampsia, virgin birth, human parthenogenesis, parthenogenesis human

Eclampsia is a life-threatening complication ocurring in pregnancy and is a leading cause of maternal and perinatal mortality. According to US study data from 1979-86 the occurance of eclampsia is reported to be 0.56 per 1,000 births comparing to pre-eclampsia which occurs in 26 per 1,000 births. Patients with eclampsia are at increased risk for pre-eclampsia-eclampsia in a later pregnancy.
Pre-eclampsia is characterized as hypertensive disorder of pregnancy.

Eclampsia and pre-eclampsia (also known as toxemia), tend to occur more commonly in first pregnancies and young mothers where it is thought that exposure to paternal antigens still has been low. Women with preexisting vascular diseases are at higher risk to develop preeclampsia and eclampsia. Such diseases are diabetes and hypertension or thrombophilic diseases such as the antiphospholipid syndrome. Conditions with a large placenta also have predisposition for toxemia. Genetic component is a risk factor as well, in sense that patients whose mother or sister had the condition are at higher risk.
Pre-eclampsia exists when a pregnant woman with gestational hypertension develops proteinuria. Originally, edema was considered part of the syndrome of pre-eclampsia, but presently the former two symptoms are sufficient for a diagnosis of pre-eclampsia.

TTags: eclampsia, human parthenogenesis, parthenogenesis, parthenogenesis human, preeclampsia, virgin birth

Tay Sachs also known as GM2 gangliosidosis is caused genetic disorder. Genes serve to direct specific development/processes within the body and are located on chromosomes. The genetic disorder in Tay Sachs disease results in the lack of an enzyme called hexosaminidase A. Gangliosides cannot be degraded without this enzyme, . Since these enzymes build up within the brain, they interfere with nerve functioning. Being recessive disorder, only people who receive two defective genes (one from the mother and one from the father) will actually have the Tay Sachs disease. People with one normal gene and one defective gene are called carriers. Since they carry the defective gene there is a high probability of passing the gene and the disease onto their offspring.
Treatment for Tay-Sachs currently only includes methods for controlling symptoms, and focus on lifestyle and care issues. Since there is no way of slowing down the progression of the disease or cure, the treatment consists of providing proper medications, nutrition, and care. Most Tay Sachs patients don’t live past the age of 4 even with treatment.
Tay-Sachs and many other defects can be diagnosed before birth by amniocentesis and chorionic villus sampling (CVS).

TTags: cystic fibrosis, sickle cell anemia, sickle cell disease, tay sachs, tay sachs disease